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Immunotherapy of auto-immune diseases by gene transfer
Immunotherapy of auto-immune diseases by gene transfer
Molecular mechanisms implicated in galectin-1-induced apoptosis: activation of the AP-1 transcription factor and downregulation of Bcl-2
“Galectin-1 induces central and peripheral cell death: implications in T-cell physiopathology”
Apoptosis as a target for gene therapy in rheumatoid arthritis
Galectins: a novel family of proteins involved in the regulation of the immune response. Implications in immunopathological processes
Activation-induced expression of CD1d antigen on mature T cells
Anti-galectin-1 autoantibodies in human Trypanosoma cruzi infection: differential expression of this beta-galactoside-binding protein in cardiac Chagas’ disease
Regulated expression of galectin-1 during B-cell activation and implications for T-cell apoptosis
Expression of galectins-1 and -3 correlates with defective mononuclear cell apoptosis in patients with juvenile idiopathic arthritis
Regulated expression and effect of galectin-1 on Trypanosoma cruzi-infected macrophages: modulation of microbicidal activity and survival
